By Syed Z. Ali MD, Yener S. Erozan MD, Ralph H. Hruban MD
Scientific and radiologic examinations can't reliably distinguish benign or inflammatory pancreatic ailment from carcinoma. The elevated use of pancreatic tremendous needle aspiration (FNA) besides advances in imaging options and the advent of endoscopic ultrasound suggestions have ended in much better detection and popularity of pancreatic plenty. accordingly, pancreatic cytopathology is indispensable to actual pre-operative prognosis but it's a tough diagnostic region with quite a few capability pitfalls and "look-alike" lesions. Skillful popularity and an expertise of the restrictions of the method are crucial in warding off misdiagnosis of those harmful lesions.
Atlas of Pancreatic Cytopathology with Histopathologic Correlations fills a void in present pathology literature. With 450 high-resolution photographs, together with photos of histopathologic and radiologic good points, this useful atlas provides an built-in method of diagnostic cytopathology that would support surgeon cytopathologists, cytotechnologists, and pathologists steer clear of strength pitfalls and "look-alike" lesions. Written through famous specialists within the box, the broad high-resolution colour photographs of the attribute positive factors of pancreatic disorder are awarded with targeted descriptions that hide vintage gains, diagnostic clues, and capability pitfalls.
Atlas of Pancreatic Cytopathology with Histopathologic Correlations is a beneficial source for the pro cytopathologist, basic and surgical pathologists, pathology trainees, and cytotechnologists.
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Extra info for Atlas of Pancreatic Cytopathology: With Histopathologic Correlations
Central areas of necrosis and calcifications are not uncommon. Pancreatic Lymphoma Although lymphoma—predominantly the nonHodgkin B-cell type—can involve peripancreatic lymph nodes and secondarily spread to the pancreas, primary lymphoma of the pancreas is uncommon. At CT, pancreatic or peripancreatic lymphoma appear as solitary or multiple hypodense masses or 15 diffuse enlargement of the gland mimicking pancreatitis. Dilatation of the pancreatic duct is uncommon—a sign helpful in differentiating lymphoma from adenocarcinoma.
This spindle cell proliferation can be so exuberant as to mimic a spindle cell neoplasm. 40 — Paraduodenal wall cyst (groove pancreatitis). A nonspecific picture with histiocytes and inflammatory cells in a mucoid background is present. No other cellular component is seen. The diagnosis can be difficult and is dependent on excluding other nonneoplastic entities (such as pseudocyst) and cystic neoplasms. 41 — Paraduodenal wall cyst (groove pancreatitis). Histiocytes and rare lymphocytes in a mucoid background are seen.
These vascular lesions should be distinguished from other forms of vasculitis. Polyarteritis nodosa strikes medium-sized muscular arteries, not the veins. 15 — Lymphoplasmacytic sclerosing pancreatitis (autoimmune pancreatitis). Immunolabeling for IgG4 highlights increased numbers (greater than 10/high-power field) of IgG4-positive plasma cells, helping to confirm the diagnosis. 16 — Hamartoma. This mass-forming lesion is composed of disorganized mature ductal and stromal elements. Hamartomas can be cystic or solid and are distinguished from chronic pancreatitis because they are localized, form a mass, and lack islets of Langerhans.