Chapter 1 fresh advancements in Flocculation exams (pages 1–10): N. F. Maclagan
Chapter 2 Lyophilic houses of Sera in Liver affliction (pages 11–21): Ch. Wunderly and F. Wuhrmann
Chapter three Amino?Acid Metabolism in Liver disorder (pages 1–10): C. E. Dent and J. M. Walshe
Chapter four adjustments within the parts of the Liver cellphone in Early Choline and Protein Deficiencies (pages 32–73): H. W. Kosterlitz
Chapter five Experimental Hepatic Cirrhosis (pages 74–80): L. E. Glynn
Chapter 6 results of Antibiotics and nutrition B12 in Cirrhosis and Necrosis of the Liver (pages 81–89): P. Gyorgy
Chapter 7 Histological stories on Fatty Infiltration of the Liver in Choline?Deficient Rats (pages 90–100): W. S. Hartroft
Chapter eight Needle Biopsy experiences of the Liver in Cirrhosis and different stipulations (pages 101–122): L. Schiff
Chapter nine The Relation of yes Mechanical elements to the creation of Ascites (pages 123–129): W. Volwiler
Chapter 10 components within the Mechanism of Ascites (pages 130–135): H. G. Kunkel
Chapter eleven The Pathogenesis of Ascites (pages 136–144): Poul Iversen
Chapter 12 Observations at the Surgical difficulties in Portal high blood pressure (pages 145–201): A. I. S Macpherson
Chapter thirteen Bile Pigment Metabolism (pages 202–210): Torben okay. With
Chapter 14 Appendix: the organic value of the Direct Diazo response (pages 211–221): Torben okay. With
Chapter 15 Relation of Liver Blood circulation to alterations in Carbohydrate Metabolism (pages 222–234): Sheila Sherlock
Chapter sixteen reports on Viral Hepatitis (pages 235–246): J. Stokes
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Extra resources for Ciba Foundation Symposium - Liver Disease
One year before : acute glomerulo-nephritis following on tonsillitis not completely cleared up combined with development of a mild LIVER DISEASE 3 CH. WUNDERLY AND F. WUHRMANN 20 nephrotic syndrome including : peripheral edema, proteinuria up t o 12 g. daily. birefringent substances in the urinary sediment, hypoproteinaemia, dysproteinaemia and lipoidaemia (see Table I). Residual nephritis with blood pressure of 150/100mm. Hg. Beginning of retinal pathological changes. No enlargement of liver, no jaundice, no enlargement of the heart ; electrocardiogram : slight damage of myocardium.
1 195 neg. 3 130 neg. Comment : Typical hemochromatosis in an alcoholic male of 58 with cirrhosis of liver of ten years’ duration. Disturbances of carbohydrate metabolism only during last year, poor response t o insulin. The disturbances in the functions of the liver are manifested notably CII. WUNDERLYAND F. WUHRMANN 18 in the metabolism of proteins and fats. The metabolism of bile pigments is only slightly affected. Cardiac condition suggestive of so-called myocardosis (Wuhrmann). The protein content of the serum as well as the affinity of serum for Evans-blue were normal (Fig.
Casein N/lOO g. 2. The lipoid P contents of liver cells for different protein N contents. weight. The phospholipin content of the liver, measured as lipoid P (LY), varies directly with the protein N content and is thus also a function of dietary protein. If the ratio LP/ DNA P is plotted against protein N/DNA P, a straight line is obtained, the origin of which passes through zero (Fig. 2). Analysis of covariance and recalculation of the LP/DNA P CHOLINE AND PROTEIN DEFICIENCIES 35 ratios for a mean N/DNA P ratio of 97 indicate that choline deficiency does not affect this relationship, while there is a significant difference between low (10 per cent) and high (40 per cent) fat diets, the latter yielding, for identical N/DNA P values, a greater LP/DNA P ratio than the former (Fig.